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L(-)-Glutathion+(oxidierte+Form)
Artikel-Nr:
(BOSSBS-0112R-A680)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-0112R-A680
Lokale Artikelnummer::
BOSSBS-0112R-A680
Beschreibung:
This gene encodes a cell surface receptor and transmembrane precursor protein that is cleaved by secretases to form a number of peptides. Some of these peptides are secreted and can bind to the acetyltransferase complex APBB1/TIP60 to promote transcriptional activation, while others form the protein basis of the amyloid plaques found in the brains of patients with Alzheimer disease. Mutations in this gene have been implicated in autosomal dominant Alzheimer disease and cerebroarterial amyloidosis (cerebral amyloid angiopathy). Multiple transcript variants encoding several different isoforms have been found for this gene.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-4227R-A680)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-4227R-A680
Lokale Artikelnummer::
BOSSBS-4227R-A680
Beschreibung:
VAPB contains 1 MSP domain and it may play a role in vesicle trafficking. Defects in VAPB are a cause of proximal adult autosomal dominant spinal muscular atrophy [MIM:182980]; also called late onset spinal muscular atrophy Finkel type. Spinal muscular atrophies are neurodegenerative disorders characterised by degeneration of lower motor neurons, leading to progressive paralysis muscular atrophy. This form is a late adult onset form of the disease (after age 20 years). The patients show a benign course, most of them remaining ambulatory 10 to 40 years after clinical onset.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-4227R)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-4227R
Lokale Artikelnummer::
BOSSBS-4227R
Beschreibung:
VAPB contains 1 MSP domain and it may play a role in vesicle trafficking. Defects in VAPB are a cause of proximal adult autosomal dominant spinal muscular atrophy [MIM:182980]; also called late onset spinal muscular atrophy Finkel type. Spinal muscular atrophies are neurodegenerative disorders characterized by degeneration of lower motor neurons, leading to progressive paralysis muscular atrophy. This form is a late adult onset form of the disease (after age 20 years). The patients show a benign course, most of them remaining ambulatory 10 to 40 years after clinical onset.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-5425R-CY3)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-5425R-CY3
Lokale Artikelnummer::
BOSSBS-5425R-CY3
Beschreibung:
MAX protein is a member of the basic helix-loop-helix leucine zipper (bHLHZ) family of transcription factors. It is able to form homodimers and heterodimers with other family members, which include Mad, Mxi1 and Myc. Myc is an oncoprotein implicated in cell proliferation, differentiation and apoptosis. The homodimers and heterodimers compete for a common DNA target site (the E box) and rearrangement among these dimer forms provides a complex system of transcriptional regulation. Mutations of this gene have been reported to be associated with hereditary pheochromocytoma. A pseudogene of this gene is located on the long arm of chromosome 7. Alternative splicing results in multiple transcript variants.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-0025M-CY5.5)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-0025M-CY5.5
Lokale Artikelnummer::
BOSSBS-0025M-CY5.5
Beschreibung:
Alzheimer's disease (AD) patients with an inherited form of the disease carry mutations in the presenilin proteins (PSEN1;PSEN2) or the amyloid precursor protein (APP). These disease-linked mutations result in increased production of the longer form of amyloid-beta (main component of amyloid deposits found in AD brains). Presenilins are postulated to regulate APP processing through their effects on gamma-secretase, an enzyme that cleaves APP. Also, it is thought that the presenilins are involved in the cleavage of the Notch receptor, such that they either directly regulate gamma-secretase activity or themselves are protease enzymes. Three alternative splice variants of PSEN1 have been identified.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-0332R-A680)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-0332R-A680
Lokale Artikelnummer::
BOSSBS-0332R-A680
Beschreibung:
Factor VIII, along with calcium and phospholipid, acts as a cofactor for factor IXa when it converts factor X to the activated form, factor Xa.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-2794R-CY7)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-2794R-CY7
Lokale Artikelnummer::
BOSSBS-2794R-CY7
Beschreibung:
Inhibits signal transduction by increasing the GTPase activity of G protein alpha subunits thereby driving them into their inactive GDP-bound form. Binds to G(i)-alpha and G(o)-alpha, but not to G(s)-alpha.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-4611R-CY7)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-4611R-CY7
Lokale Artikelnummer::
BOSSBS-4611R-CY7
Beschreibung:
Forms a water-specific channel that provides the plasma membranes of renal collecting duct with high permeability to water, thereby permitting water to move in the direction of an osmotic gradient.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-4043R)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-4043R
Lokale Artikelnummer::
BOSSBS-4043R
Beschreibung:
UGP2 (UDP glucose pyrophosphorylase 2) is an important intermediary in cellular metabolic pathways. It transfers a glucose moiety from glucose-1-phosphate to MgUTP and forms UDP glucose and MgPPi.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-2794R-CY5)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-2794R-CY5
Lokale Artikelnummer::
BOSSBS-2794R-CY5
Beschreibung:
Inhibits signal transduction by increasing the GTPase activity of G protein alpha subunits thereby driving them into their inactive GDP-bound form. Binds to G(i)-alpha and G(o)-alpha, but not to G(s)-alpha.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-15086R-A750)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-15086R-A750
Lokale Artikelnummer::
BOSSBS-15086R-A750
Beschreibung:
C1r B chain is a serine protease that combines with C1q and C1s to form C1, the first component of the classical pathway of the complement system.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-1127R-CY7)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-1127R-CY7
Lokale Artikelnummer::
BOSSBS-1127R-CY7
Beschreibung:
Component of the sarcoglycan complex, a subcomplex of the dystrophin-glycoprotein complex which forms a link between the F-actin cytoskeleton and the extracellular matrix.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-0300R-CY3)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-0300R-CY3
Lokale Artikelnummer::
BOSSBS-0300R-CY3
Beschreibung:
Membrane-anchored forms may play a role in cellular adhesion. Megakaryocyte-potentiating factor (MPF) potentiates megakaryocyte colony formation in vitro.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-7166R-FITC)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-7166R-FITC
Lokale Artikelnummer::
BOSSBS-7166R-FITC
Beschreibung:
Gel-forming glycoprotein of gastric and respiratoy tract epithelia that protects the mucosa from infection and chemical damage by binding to inhaled microrganisms and particles that are subsequently removed by the mucocilary system.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-1398R-A680)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-1398R-A680
Lokale Artikelnummer::
BOSSBS-1398R-A680
Beschreibung:
Stimulates transcription. Binds to purine-rich DNA sequences. Can form a ternary complex with the serum response factor and the ETS and SRF motifs of the fos serum response element.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-2668R-A555)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-2668R-A555
Lokale Artikelnummer::
BOSSBS-2668R-A555
Beschreibung:
Binds tightly to hydroxyapatite. Appears to form an integral part of the mineralized matrix. Probably important to cell-matrix interaction. Promotes Arg-Gly-Asp-dependent cell attachment.
VE:
1 * 100 µl
Preis auf Anfrage
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 noch angezeigt wird und Sie Hilfe benötigen, rufen Sie uns an 1-800-932 - 5000.
Lager für diesen Artikel ist begrenzt, kann aber in einem Lagerhaus in Ihrer Nähe zur Verfügung. Bitte stellen Sie sicher, dass Sie in sind angemeldet auf dieser Seite, so dass verfügbare Bestand angezeigt werden können. Wenn das
noch angezeigt wird und Sie Hilfe benötigen, rufen Sie uns an 1-800-932 - 5000.
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