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(1-(Pyridin-2-yl)cyclohexyl)methanamine+tosylate+salt
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(1-(Pyridin-2-yl)cyclohexyl)methanamine+tosylate+salt
Lieferant:
Sigma-Aldrich
Beschreibung:
2-(2-Hydroxyethyl)pyridin, Sigma-Aldrich®
Lieferant:
MP Biomedicals
Beschreibung:
L-Ascorbic Acid, Sodium Salt is a vitamin widely distributed in the plant and animal kingdom.
Artikel-Nr:
(SERA11064.02)
Lieferant:
Serva
Hersteller-Artikelnummer::
11064.02
Lokale Artikelnummer::
SERA11064.02
Beschreibung:
L(-)-Äpfelsäure Mononatriumsalz
VE:
1 * 25 g
 This is a MarketSource item. Additional charges may apply
Lieferant:
Sigma-Aldrich
Beschreibung:
Pyridin-2-carbaldehyd, Sigma-Aldrich®
Lieferant:
Thermo Scientific
Beschreibung:
3-(Aminomethyl)pyridin
Artikel-Nr:
(MOLEM17692553)
Lieferant:
Molekula
Hersteller-Artikelnummer::
M17692553
Lokale Artikelnummer::
MOLEM17692553
Beschreibung:
Acridinorange (Zinkchlorid-Doppelsalz)
VE:
1 * 25 g
This is a MarketSource item. Additional charges may apply
Artikel-Nr:
(SIAL2854-15ML)
Lieferant:
Sigma-Aldrich
Hersteller-Artikelnummer::
2854-15ML
Lokale Artikelnummer::
SIAL2854-15ML
Beschreibung:
EDTA Dinatriumsalz 2% in wässriger Lösung, Sigma-Aldrich®
VE:
1 * 15 mL
Lieferant:
Sigma-Aldrich
Beschreibung:
2'-Desoxycytidin-5'-triphosphorsäure Dinatriumsalz (dCTP-Na2), Sigma-Aldrich®
Artikel-Nr:
(BOSSBS-13627R-A647)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-13627R-A647
Lokale Artikelnummer::
BOSSBS-13627R-A647
Beschreibung:
The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport, and maintain cellular volume. CLC-KA is a kidney-specific chloride channel that mediates transepithelial chloride transport in the thin ascending limb of the Henle loop in the inner medulla. CLC-KA plays a crucial role in urine concentration. The gene encoding human CLC-KA maps to chromosome 1p36. Mutations in this gene may be associated with nephrogenic diabetes insipidus in those cases where mutations in the vasopressin V2 receptor and the AQP2 water channel are lacking. CLC-KB mediates basolateral chloride ion efflux in the thick ascending limb and in more distal nephron segments. The gene encoding human CLC-KB maps to chromosome 1p36. Mutations in this gene cause type III Barter’s syndrome which is characterized by renal salt-wasting and low blood pressure.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-13627R-A350)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-13627R-A350
Lokale Artikelnummer::
BOSSBS-13627R-A350
Beschreibung:
The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport, and maintain cellular volume. CLC-KA is a kidney-specific chloride channel that mediates transepithelial chloride transport in the thin ascending limb of the Henle loop in the inner medulla. CLC-KA plays a crucial role in urine concentration. The gene encoding human CLC-KA maps to chromosome 1p36. Mutations in this gene may be associated with nephrogenic diabetes insipidus in those cases where mutations in the vasopressin V2 receptor and the AQP2 water channel are lacking. CLC-KB mediates basolateral chloride ion efflux in the thick ascending limb and in more distal nephron segments. The gene encoding human CLC-KB maps to chromosome 1p36. Mutations in this gene cause type III Barter’s syndrome which is characterized by renal salt-wasting and low blood pressure.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-13627R-HRP)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-13627R-HRP
Lokale Artikelnummer::
BOSSBS-13627R-HRP
Beschreibung:
The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport, and maintain cellular volume. CLC-KA is a kidney-specific chloride channel that mediates transepithelial chloride transport in the thin ascending limb of the Henle loop in the inner medulla. CLC-KA plays a crucial role in urine concentration. The gene encoding human CLC-KA maps to chromosome 1p36. Mutations in this gene may be associated with nephrogenic diabetes insipidus in those cases where mutations in the vasopressin V2 receptor and the AQP2 water channel are lacking. CLC-KB mediates basolateral chloride ion efflux in the thick ascending limb and in more distal nephron segments. The gene encoding human CLC-KB maps to chromosome 1p36. Mutations in this gene cause type III Barter’s syndrome which is characterized by renal salt-wasting and low blood pressure.
VE:
1 * 100 µl
Artikel-Nr:
(PROOMM0432.01)
Lieferant:
LGC Standards PROMOCHEM
Hersteller-Artikelnummer::
MM0432.01
Lokale Artikelnummer::
PROOMM0432.01
Beschreibung:
Propyl-4-hydroxybenzoat Natriumsalz
VE:
1 * 100 mg
Artikel-Nr:
(BOSSBS-10307R-A350)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-10307R-A350
Lokale Artikelnummer::
BOSSBS-10307R-A350
Beschreibung:
The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport and maintain cellular volume. The genes encoding human CLC-1 through CLC-7 map to chromosomes 7q32, 3q28, 4q32, Xp22.3, Xp11.23-p11.22, 1p36 and 16p13, respectively. CLC1 is highly expressed in skeletal muscle. Mutations in the gene encoding CLC1 lead to myotonia, an inheritable disorder characterized by muscle stiffness and renal salt wasting. CLC2 is highly expressed in the epithelia of several organs including lung, which suggests CLC2 may be a possible therapeutic target for cystic fibrosis. CLC3 expression is particularly abundant in neuronal tissue, while CLC4 expression is evident in skeletal and cardiac muscle as well as brain. Mutations in the gene encoding CLC5 lead to Dent’s disease, a renal disorder characterized by proteinuria and hypercalciuria. CLC6 and CLC7 are broadly expressed in several tissues including testis, kidney, brain and muscle.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-10307R-A750)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-10307R-A750
Lokale Artikelnummer::
BOSSBS-10307R-A750
Beschreibung:
The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport and maintain cellular volume. The genes encoding human CLC-1 through CLC-7 map to chromosomes 7q32, 3q28, 4q32, Xp22.3, Xp11.23-p11.22, 1p36 and 16p13, respectively. CLC1 is highly expressed in skeletal muscle. Mutations in the gene encoding CLC1 lead to myotonia, an inheritable disorder characterised by muscle stiffness and renal salt wasting. CLC2 is highly expressed in the epithelia of several organs including lung, which suggests CLC2 may be a possible therapeutic target for cystic fibrosis. CLC3 expression is particularly abundant in neuronal tissue, while CLC4 expression is evident in skeletal and cardiac muscle as well as brain. Mutations in the gene encoding CLC5 lead to Dent's disease, a renal disorder characterised by proteinuria and hypercalciuria. CLC6 and CLC7 are broadly expressed in several tissues including testis, kidney, brain and muscle.
VE:
1 * 100 µl
Lieferant:
Sigma-Aldrich
Beschreibung:
Lithiumdodecylsulfat, Sigma-Aldrich®
Artikel-Nr:
(MOLEM79277079)
Lieferant:
Molekula
Hersteller-Artikelnummer::
M79277079
Lokale Artikelnummer::
MOLEM79277079
Beschreibung:
Adenosin-5'-monophosphat Dinatriumsalz (AMP Dinatriumsalz) Hydrat
VE:
1 * 25 g
This is a MarketSource item. Additional charges may apply
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 noch angezeigt wird und Sie Hilfe benötigen, rufen Sie uns an 1-800-932 - 5000.
Lager für diesen Artikel ist begrenzt, kann aber in einem Lagerhaus in Ihrer Nähe zur Verfügung. Bitte stellen Sie sicher, dass Sie in sind angemeldet auf dieser Seite, so dass verfügbare Bestand angezeigt werden können. Wenn das
noch angezeigt wird und Sie Hilfe benötigen, rufen Sie uns an 1-800-932 - 5000.
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