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(+\/-)-3-(trans-2-(Trifluoromethyl)cyclopropyl)benzoic+acid


151 099  results were found

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Lieferant:  APOLLO SCIENTIFIC
Beschreibung:   3-Amino-2-methylphenyl trifluoromethanesulphonate
Artikel-Nr: (EHERC13405000)

Lieferant:  EHRENSTORFER
Hersteller-Artikelnummer:: C13405000
Lokale Artikelnummer:: EHERC13405000
Beschreibung:   Farnesol (Isomerengemisch)
VE:  1 * 0,25 g
Artikel-Nr: (BLDPBD102760-1G)

Lieferant:  BLD PHARMATECH GMBH
Hersteller-Artikelnummer:: BD102760-1G
Lokale Artikelnummer:: BLDPBD102760-1G
Beschreibung:   5-Brom-2-iodbenzotrifluorid 96%
VE:  1 * 1 g
Lieferant:  Alfa Aesar
Beschreibung:   4-Brom-α,α,α,2,3,5,6-heptafluortoluol ≥99%
Lieferant:  APOLLO SCIENTIFIC
Beschreibung:   2-Fluoro-5-trifluoromethylbenzyl chloride
Artikel-Nr: (BOSSBS-3703R-CY5)

Lieferant:  Bioss
Hersteller-Artikelnummer:: BS-3703R-CY5
Lokale Artikelnummer:: BOSSBS-3703R-CY5
Beschreibung:   Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. In cooperation with mitochondrial PPIF is involved in activating oxidative stress-induced necrosis; the function is largely independent of transcription. Induces the transcription of long intergenic non-coding RNA p21 (lincRNA-p21) and lincRNA-Mkln1. LincRNA-p21 participates in TP53-dependent transcriptional repression leading to apoptosis and seem to have to effect on cell-cycle regulation. Implicated in Notch signaling cross-over. Prevents CDK7 kinase activity when associated to CAK complex in response to DNA damage, thus stopping cell cycle progression. Isoform 2 enhances the transactivation activity of isoform 1 from some but not all TP53-inducible promoters. Isoform 4 suppresses transactivation activity and impairs growth suppression mediated by isoform 1. Isoform 7 inhibits isoform 1-mediated apoptosis.
VE:  1 * 100 µl
Artikel-Nr: (BOSSBS-3703R-CY5.5)

Lieferant:  Bioss
Hersteller-Artikelnummer:: BS-3703R-CY5.5
Lokale Artikelnummer:: BOSSBS-3703R-CY5.5
Beschreibung:   Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. In cooperation with mitochondrial PPIF is involved in activating oxidative stress-induced necrosis; the function is largely independent of transcription. Induces the transcription of long intergenic non-coding RNA p21 (lincRNA-p21) and lincRNA-Mkln1. LincRNA-p21 participates in TP53-dependent transcriptional repression leading to apoptosis and seem to have to effect on cell-cycle regulation. Implicated in Notch signaling cross-over. Prevents CDK7 kinase activity when associated to CAK complex in response to DNA damage, thus stopping cell cycle progression. Isoform 2 enhances the transactivation activity of isoform 1 from some but not all TP53-inducible promoters. Isoform 4 suppresses transactivation activity and impairs growth suppression mediated by isoform 1. Isoform 7 inhibits isoform 1-mediated apoptosis.
VE:  1 * 100 µl
Artikel-Nr: (EHERC10390000)

Lieferant:  EHRENSTORFER
Hersteller-Artikelnummer:: C10390000
Lokale Artikelnummer:: EHERC10390000
Beschreibung:   Azobenzol
VE:  1 * 0,5 g
Artikel-Nr: (MOLEM57735199)

Lieferant:  Molekula
Hersteller-Artikelnummer:: M57735199
Lokale Artikelnummer:: MOLEM57735199
Beschreibung:   Resveratrol
VE:  1 * 1 g
Market Source Item This is a MarketSource item. Additional charges may apply
Artikel-Nr: (BOSSBS-1650R-CY7)

Lieferant:  Bioss
Hersteller-Artikelnummer:: BS-1650R-CY7
Lokale Artikelnummer:: BOSSBS-1650R-CY7
Beschreibung:   Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. In cooperation with mitochondrial PPIF is involved in activating oxidative stress-induced necrosis; the function is largely independent of transcription. Induces the transcription of long intergenic non-coding RNA p21 (lincRNA-p21) and lincRNA-Mkln1. LincRNA-p21 participates in TP53-dependent transcriptional repression leading to apoptosis and seem to have to effect on cell-cycle regulation. Implicated in Notch signaling cross-over. Prevents CDK7 kinase activity when associated to CAK complex in response to DNA damage, thus stopping cell cycle progression. Isoform 2 enhances the transactivation activity of isoform 1 from some but not all TP53-inducible promoters. Isoform 4 suppresses transactivation activity and impairs growth suppression mediated by isoform 1. Isoform 7 inhibits isoform 1-mediated apoptosis.
VE:  1 * 100 µl
Artikel-Nr: (BOSSBS-3705R-CY5)

Lieferant:  Bioss
Hersteller-Artikelnummer:: BS-3705R-CY5
Lokale Artikelnummer:: BOSSBS-3705R-CY5
Beschreibung:   Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. In cooperation with mitochondrial PPIF is involved in activating oxidative stress-induced necrosis; the function is largely independent of transcription. Induces the transcription of long intergenic non-coding RNA p21 (lincRNA-p21) and lincRNA-Mkln1. LincRNA-p21 participates in TP53-dependent transcriptional repression leading to apoptosis and seem to have to effect on cell-cycle regulation. Implicated in Notch signaling cross-over. Prevents CDK7 kinase activity when associated to CAK complex in response to DNA damage, thus stopping cell cycle progression. Isoform 2 enhances the transactivation activity of isoform 1 from some but not all TP53-inducible promoters. Isoform 4 suppresses transactivation activity and impairs growth suppression mediated by isoform 1. Isoform 7 inhibits isoform 1-mediated apoptosis.
VE:  1 * 100 µl
Artikel-Nr: (SPCMV1257-25KGBL)

Lieferant:  Spectrum Chemical
Hersteller-Artikelnummer:: V1257-25KGBL
Lokale Artikelnummer:: SPCMV1257-25KGBL
Beschreibung:   BSE/TSE statement (available upon request)
Allergen statement (available upon request)
VE:  1 * 25 kg
Lieferant:  APOLLO SCIENTIFIC
Beschreibung:   2H-Perfluoro-5,8,11-trimethyl-3,6,9,12-tetraoxatetradecane 97%
Artikel-Nr: (BOSSBS-3705R-CY3)

Lieferant:  Bioss
Hersteller-Artikelnummer:: BS-3705R-CY3
Lokale Artikelnummer:: BOSSBS-3705R-CY3
Beschreibung:   Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. In cooperation with mitochondrial PPIF is involved in activating oxidative stress-induced necrosis; the function is largely independent of transcription. Induces the transcription of long intergenic non-coding RNA p21 (lincRNA-p21) and lincRNA-Mkln1. LincRNA-p21 participates in TP53-dependent transcriptional repression leading to apoptosis and seem to have to effect on cell-cycle regulation. Implicated in Notch signaling cross-over. Prevents CDK7 kinase activity when associated to CAK complex in response to DNA damage, thus stopping cell cycle progression. Isoform 2 enhances the transactivation activity of isoform 1 from some but not all TP53-inducible promoters. Isoform 4 suppresses transactivation activity and impairs growth suppression mediated by isoform 1. Isoform 7 inhibits isoform 1-mediated apoptosis.
VE:  1 * 100 µl
Artikel-Nr: (BOSSBS-3705R-CY7)

Lieferant:  Bioss
Hersteller-Artikelnummer:: BS-3705R-CY7
Lokale Artikelnummer:: BOSSBS-3705R-CY7
Beschreibung:   Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. In cooperation with mitochondrial PPIF is involved in activating oxidative stress-induced necrosis; the function is largely independent of transcription. Induces the transcription of long intergenic non-coding RNA p21 (lincRNA-p21) and lincRNA-Mkln1. LincRNA-p21 participates in TP53-dependent transcriptional repression leading to apoptosis and seem to have to effect on cell-cycle regulation. Implicated in Notch signaling cross-over. Prevents CDK7 kinase activity when associated to CAK complex in response to DNA damage, thus stopping cell cycle progression. Isoform 2 enhances the transactivation activity of isoform 1 from some but not all TP53-inducible promoters. Isoform 4 suppresses transactivation activity and impairs growth suppression mediated by isoform 1. Isoform 7 inhibits isoform 1-mediated apoptosis.
VE:  1 * 100 µl
Artikel-Nr: (BOSSBS-3703R-CY7)

Lieferant:  Bioss
Hersteller-Artikelnummer:: BS-3703R-CY7
Lokale Artikelnummer:: BOSSBS-3703R-CY7
Beschreibung:   Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. In cooperation with mitochondrial PPIF is involved in activating oxidative stress-induced necrosis; the function is largely independent of transcription. Induces the transcription of long intergenic non-coding RNA p21 (lincRNA-p21) and lincRNA-Mkln1. LincRNA-p21 participates in TP53-dependent transcriptional repression leading to apoptosis and seem to have to effect on cell-cycle regulation. Implicated in Notch signaling cross-over. Prevents CDK7 kinase activity when associated to CAK complex in response to DNA damage, thus stopping cell cycle progression. Isoform 2 enhances the transactivation activity of isoform 1 from some but not all TP53-inducible promoters. Isoform 4 suppresses transactivation activity and impairs growth suppression mediated by isoform 1. Isoform 7 inhibits isoform 1-mediated apoptosis.
VE:  1 * 100 µl
Preis auf Anfrage
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Lager für diesen Artikel ist begrenzt, kann aber in einem Lagerhaus in Ihrer Nähe zur Verfügung. Bitte stellen Sie sicher, dass Sie in sind angemeldet auf dieser Seite, so dass verfügbare Bestand angezeigt werden können. Wenn das call noch angezeigt wird und Sie Hilfe benötigen, rufen Sie uns an 1-800-932 - 5000.
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