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6-Formylpicolinonitrile
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6-Formylpicolinonitrile
Artikel-Nr:
(BOSSBS-5517R-A350)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-5517R-A350
Lokale Artikelnummer::
BOSSBS-5517R-A350
Beschreibung:
NFKB1 or NFKB2 is bound to REL, RELA, or RELB to form the NFKB complex. The NFKB complex is inhibited by I-kappa-B proteins (NFKBIA, or NFKBIB), which inactivate NF-kappa-B by trapping it in the cytoplasm. Phosphorylation of serine residues on the I-kappa-B proteins by kinases (IKBKA, or IKBKB) marks them for destruction via the ubiquitination pathway, thereby allowing activation of the NF-kappa-B complex. Activated NFKB complex translocates into the nucleus and binds DNA at kappa-B-binding motifs such as 5-prime GGGRNNYYCC 3-prime or 5-prime HGGARNYYCC 3-prime (where H is A, C, or T; R is an A or G purine; and Y is a C or T pyrimidine). For some genes, activation requires NFKB interaction with other transcription factors, such as STAT, AP1 (JUN), and NFAT.
VE:
1 * 100 µl
Artikel-Nr:
(472681000.)
Lieferant:
Thermo Scientific
Hersteller-Artikelnummer::
472681000
Lokale Artikelnummer::
ACRO472681000
Beschreibung:
4-((4-(Mesitylamino)pyrimidin-2-yl)amino)benzonitrile 98%
VE:
1 * 100 mg
New Product
Artikel-Nr:
(BOSSBS-5517R-A680)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-5517R-A680
Lokale Artikelnummer::
BOSSBS-5517R-A680
Beschreibung:
NFKB1 or NFKB2 is bound to REL, RELA, or RELB to form the NFKB complex. The NFKB complex is inhibited by I-kappa-B proteins (NFKBIA, or NFKBIB), which inactivate NF-kappa-B by trapping it in the cytoplasm. Phosphorylation of serine residues on the I-kappa-B proteins by kinases (IKBKA, or IKBKB) marks them for destruction via the ubiquitination pathway, thereby allowing activation of the NF-kappa-B complex. Activated NFKB complex translocates into the nucleus and binds DNA at kappa-B-binding motifs such as 5-prime GGGRNNYYCC 3-prime or 5-prime HGGARNYYCC 3-prime (where H is A, C, or T; R is an A or G purine; and Y is a C or T pyrimidine). For some genes, activation requires NFKB interaction with other transcription factors, such as STAT, AP1 (JUN), and NFAT.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-5517R-A647)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-5517R-A647
Lokale Artikelnummer::
BOSSBS-5517R-A647
Beschreibung:
NFKB1 or NFKB2 is bound to REL, RELA, or RELB to form the NFKB complex. The NFKB complex is inhibited by I-kappa-B proteins (NFKBIA, or NFKBIB), which inactivate NF-kappa-B by trapping it in the cytoplasm. Phosphorylation of serine residues on the I-kappa-B proteins by kinases (IKBKA, or IKBKB) marks them for destruction via the ubiquitination pathway, thereby allowing activation of the NF-kappa-B complex. Activated NFKB complex translocates into the nucleus and binds DNA at kappa-B-binding motifs such as 5-prime GGGRNNYYCC 3-prime or 5-prime HGGARNYYCC 3-prime (where H is A, C, or T; R is an A or G purine; and Y is a C or T pyrimidine). For some genes, activation requires NFKB interaction with other transcription factors, such as STAT, AP1 (JUN), and NFAT.
VE:
1 * 100 µl
Artikel-Nr:
(COBBST-8295-1G)
Lieferant:
COMBI-BLOCKS
Hersteller-Artikelnummer::
ST-8295-1G
Lokale Artikelnummer::
COBBST-8295-1G
Beschreibung:
5-Chlor-2-methyl-7H-isoxazolo[2,3-a]pyrimidin-7-on
VE:
1 * 1 g
 This is a MarketSource item. Additional charges may apply
Artikel-Nr:
(BOSSBS-5517R-A488)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-5517R-A488
Lokale Artikelnummer::
BOSSBS-5517R-A488
Beschreibung:
NFKB1 or NFKB2 is bound to REL, RELA, or RELB to form the NFKB complex. The NFKB complex is inhibited by I-kappa-B proteins (NFKBIA, or NFKBIB), which inactivate NF-kappa-B by trapping it in the cytoplasm. Phosphorylation of serine residues on the I-kappa-B proteins by kinases (IKBKA, or IKBKB) marks them for destruction via the ubiquitination pathway, thereby allowing activation of the NF-kappa-B complex. Activated NFKB complex translocates into the nucleus and binds DNA at kappa-B-binding motifs such as 5-prime GGGRNNYYCC 3-prime or 5-prime HGGARNYYCC 3-prime (where H is A, C, or T; R is an A or G purine; and Y is a C or T pyrimidine). For some genes, activation requires NFKB interaction with other transcription factors, such as STAT, AP1 (JUN), and NFAT.
VE:
1 * 100 µl
Artikel-Nr:
(BOSSBS-5517R-A750)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-5517R-A750
Lokale Artikelnummer::
BOSSBS-5517R-A750
Beschreibung:
NFKB1 or NFKB2 is bound to REL, RELA, or RELB to form the NFKB complex. The NFKB complex is inhibited by I-kappa-B proteins (NFKBIA, or NFKBIB), which inactivate NF-kappa-B by trapping it in the cytoplasm. Phosphorylation of serine residues on the I-kappa-B proteins by kinases (IKBKA, or IKBKB) marks them for destruction via the ubiquitination pathway, thereby allowing activation of the NF-kappa-B complex. Activated NFKB complex translocates into the nucleus and binds DNA at kappa-B-binding motifs such as 5-prime GGGRNNYYCC 3-prime or 5-prime HGGARNYYCC 3-prime (where H is A, C, or T; R is an A or G purine; and Y is a C or T pyrimidine). For some genes, activation requires NFKB interaction with other transcription factors, such as STAT, AP1 (JUN), and NFAT.
VE:
1 * 100 µl
Lieferant:
Cayman Chemical
Beschreibung:
WYE-125132 is an ATP-competitive inhibitor of mTOR (IC<sub>50</sub> = 0,19 nM) that inhibits signaling through both mTORC1 and mTORC2. It is selective for mTOR over phosphatidylinositol 3-kinase isoforms. WYE-125132 is effective against mTORC1 and mTORC2 in diverse cancer models, both <i>in vitro</i> and <i>in vivo</i>. Oral administration of WYE-125132 alone blocks mTOR signaling and prevents tumor growth in breast, lung, renal, and glioma cancer xenografts in mice, while combination therapy with the VEGF-inhibitor bevacizumab causes complete regression of A498 renal carcinoma tumors. In addition to its applications in cancer, WYE-125132 has been used to delineate novel aspects of mTOR signaling.
Artikel-Nr:
(ENAMEN300-14372.1G)
Lieferant:
SIA ENAMINE
Hersteller-Artikelnummer::
EN300-14372.1G
Lokale Artikelnummer::
ENAMEN300-14372.1G
Beschreibung:
5-Chlor-2-methyl-7H-isoxazolo[2,3-a]pyrimidin-7-on
VE:
1 * 1 g
This is a MarketSource item. Additional charges may apply
Artikel-Nr:
(BOSSBS-5517R-HRP)
Lieferant:
Bioss
Hersteller-Artikelnummer::
BS-5517R-HRP
Lokale Artikelnummer::
BOSSBS-5517R-HRP
Beschreibung:
NFKB1 or NFKB2 is bound to REL, RELA, or RELB to form the NFKB complex. The NFKB complex is inhibited by I-kappa-B proteins (NFKBIA, or NFKBIB), which inactivate NF-kappa-B by trapping it in the cytoplasm. Phosphorylation of serine residues on the I-kappa-B proteins by kinases (IKBKA, or IKBKB) marks them for destruction via the ubiquitination pathway, thereby allowing activation of the NF-kappa-B complex. Activated NFKB complex translocates into the nucleus and binds DNA at kappa-B-binding motifs such as 5-prime GGGRNNYYCC 3-prime or 5-prime HGGARNYYCC 3-prime (where H is A, C, or T; R is an A or G purine; and Y is a C or T pyrimidine). For some genes, activation requires NFKB interaction with other transcription factors, such as STAT, AP1 (JUN), and NFAT.
VE:
1 * 100 µl
Lieferant:
Sigma-Aldrich
Beschreibung:
1,5-Diphenylcarbazid, Supelco®
Artikel-Nr:
(42871-10G)
Lieferant:
Sigma-Aldrich
Hersteller-Artikelnummer::
42871-10G
Lokale Artikelnummer::
SUPL42871-10G
Beschreibung:
1,5-Diphenylcarbazon, Supelco®
VE:
1 * 10 g
Lieferant:
Merck
Beschreibung:
1,5-Pentandiol zur Synthese, Sigma-Aldrich®
Lieferant:
Merck
Beschreibung:
1,5-Naphthalindisulfonsäure zur Synthese, Sigma-Aldrich®
Lieferant:
Merck
Beschreibung:
1,5-Diphenylcarbazid ACS, Reag. Ph. Eur. analytisches Reagens, Supelco®
Lieferant:
Merck
Beschreibung:
Ethyl lactate is a safer and more sustainable alternative to ethyl acetate and acetone. It is an ester of natural L-lactic acid, which is produced by fermentation of sugar.
Preis auf Anfrage
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 noch angezeigt wird und Sie Hilfe benötigen, rufen Sie uns an 1-800-932 - 5000.
Lager für diesen Artikel ist begrenzt, kann aber in einem Lagerhaus in Ihrer Nähe zur Verfügung. Bitte stellen Sie sicher, dass Sie in sind angemeldet auf dieser Seite, so dass verfügbare Bestand angezeigt werden können. Wenn das
noch angezeigt wird und Sie Hilfe benötigen, rufen Sie uns an 1-800-932 - 5000.
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